Articles Vol. 67 No. CĐ6-NCKH 16/06/2026

CLINICAL AND LABORATORY CHARACTERISTICS AND TREATMENT OUTCOMES OF CYTOMEGALOVIRUS-ASSOCIATED THROMBOCYTOPENIA AT THE NATIONAL CHILDREN’S HOSPITAL

Nguyen Van Trung1,2, Nguyen Hoang Nam3,4, Nguyen Thi Huong Mai1,2
1 Hanoi Medical University
2 Trường Đại học Y Hà Nội
3 National Children's Hospital
4 Bệnh viện Nhi Trung ương
DOI: 10.52163/yhc.v67iCD6.5294
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Abstract

Objective: To describe the clinical and laboratory characteristics and the treatment outcomes of patients with cytomegalovirus associated thrombocytopenia at the Clinical Hematology Department - National Children’s Hospital.

Subjects: Seventy-six patients under 18 years old diagnosed with CMV-associated thrombocytopenia and treated at the Clinical Hematology Department - National Children’s Hospital, of whom treatment outcomes were evaluated in 74 patients who were followed up during treatment.

Methods: Descriptive case series.

Results: CMV associated thrombocytopenia in this study was mostly observed in children under 12 months of age, accounting for 80.3%. Males were more common than females. The most common symptom was bleeding (100%), anemia (53.9%). Other symptoms were also recorded such as diarrhea (30.3%) and respiratory tract infection (22.3%). Most patients had severe thrombocytopenia <20 G/L (88.2%). The CMV viral load in blood was inversely proportional to the platelet count. Clinical course types included: acute (82.4%), persistent (10.8%), and chronic (6.8%). Complete, partial, and no response rates to treatment were 55.4%, 43.2%, and 1.4%, respectively. Ten patients with CMV-associated thrombocytopenia had complicated clinical manifestations (hepatitis, bronchitis, fever, etc.) and did not respond to initial treatment with corticosteroids, intravenous immunoglobulin; after ganciclovir was added to the regimen, 9 out of 10 patients responded to treatment.

Conclusion: Patients with CMV-associated thrombocytopenia were mainly infants under 12 months of age, with the most common clinical manifestation being bleeding, followed by anemia. Most patients had severe thrombocytopenia (<20 G/L) and the platelet count decreased as the viral load increased. Antiviral therapy should be added for patients who do not respond to standard immune thrombocytopenia therapy.

References
[1]
Shimanovsky A, Patel D, Wasser J. Refractory Immune Thrombocytopenic Purpura and Cytomegalovirus Infection: A Call for a Change in the Current Guidelines. Mediterr J Hematol Infect Dis. 2016;8(1):e2016010. doi:10.4084/MJHID.2016.010. Google Scholar
[2]
Braga JAP, Loggetto SR, Hoepers AT de C et al. Guidelines on the diagnosis of primary immune thrombocytopenia in children and adolescents. Rev Bras Hematol E Hemoter. 2013;35(5):358-365. doi:10.5581/1516-8484.20130105 Google Scholar
[3]
Eisenberg MJ, Kaplan B. Cytomegalovirus-induced thrombocytopenia in an immunocompetent adult. West J Med. 1993;158(5):525-526. Google Scholar
[4]
Rodeghiero F, Stasi R, Gernsheimer T et al. Standardization of terminology, definitions and outcome criteria in immune thrombocytopenic purpura of adults and children: report from an international working group. Blood 2009; 113: 2386-2393. doi: 10.1182/blood-2008-07-162503 Google Scholar
[5]
Kang JW, Kim GN, Kim SY, et al. Clinical and radiologic evaluation of cytomegalovirus-induced thrombocytopenia in infants between 1 and 6 months of age. Korean J Hematol. 2010;45(1):29-35. doi:10.5045/kjh. 2010.45.1.29 Google Scholar
[6]
Khôi TQ, Mậu NK, Trí VĐ và cộng sự. Đặc điểm lâm sàng, cận lâm sàng và kết quả điều trị nhiễm Cytomegalovirus ở trẻ sơ sinh tại Bệnh viện Nhi Đồng 1. Tạp Chí Học Việt Nam. 2025;554(2). doi:10.51298/vmj.v554i2.15765 Google Scholar
[7]
Levy AS, Bussel J. Immune thrombocytopenic purpura: investigation of the role of cytomegalovirus infection. Br J Haematol. 2004;126(4):622-623. doi:10.1111/j.1365-2141. 2004.05068.x Google Scholar
[8]
Zafar H, Anwar S, Faizan M et al. Clinical features and outcome in paediatric newly diagnosed immune thrombocytopenic purpura in a tertiary care centre. J Pak Med Assoc. 2018;68(10):1474-1478. doi:10.5455/JPMA.272401. PMID: 30344575; PMCID: PMC6191794. Google Scholar